Translational Neurodegeneration is an open access, peer-reviewed journal that covers research, therapeutics and education for all aspects of neurodegenerative diseases. By offering a high-visibility forum for new insights and discussions, Translational Neurodegeneration creates a novel interface between the fields of basic, translational, and clinical research. It includes Parkinson's disease and movement disorders, Alzheimer's disease and other dementias, motor neuron disease and other neurodegenerative diseases.
翻译神经退行性变是一个开放获取，同行评审的期刊，涵盖研究，治疗和教育的所有方面的神经退行性疾病。通过为新的见解和讨论提供一个高知名度的论坛，转化神经变性创造了一个基础，转化和临床研究领域之间的新界面。它包括帕金森氏病和运动障碍、阿尔茨海默氏病和其他痴呆、运动神经元疾病和其他神经退行性疾病。
The interplay of aging, genetics and environmental factors in the pathogenesis of Parkinson’s disease
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0165-9
Blocking meningeal lymphatic drainage aggravates Parkinson’s disease-like pathology in mice overexpressing mutated α-synuclein
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0147-y
Exploratory study on microRNA profiles from plasma-derived extracellular vesicles in Alzheimer’s disease and dementia with Lewy bodies
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0169-5
Beyond the synucleinopathies: alpha synuclein as a driving force in neurodegenerative comorbidities
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0172-x
Efficient RT-QuIC seeding activity for α-synuclein in olfactory mucosa samples of patients with Parkinson’s disease and multiple system atrophy
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0164-x
Current progress of mitochondrial transplantation that promotes neuronal regeneration
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0158-8
Constipation, deficit in colon contractions and alpha-synuclein inclusions within the colon precede motor abnormalities and neurodegeneration in the central nervous system in a mouse model of alpha-synucleinopathy
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0146-z
Parkinson’s disease in China: a forty-year growing track of bedside work
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0162-z
Erythrocytic α-Synuclein as a potential biomarker for Parkinson’s disease
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0155-y
Microbiome changes: an indicator of Parkinson’s disease?
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0175-7
Targeting γ-secretase triggers the selective enrichment of oligomeric APP-CTFs in brain extracellular vesicles from Alzheimer cell and mouse models
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0176-6
Detecting neurodegenerative pathology in multiple sclerosis before irreversible brain tissue loss sets in
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0178-4
Longitudinal diffusion tensor magnetic resonance imaging analysis at the cohort level reveals disturbed cortical and callosal microstructure with spared corticospinal tract in the TDP-43G298S ALS mouse model
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0163-y
A longitudinal assessment of retinal function and structure in the APP/PS1 transgenic mouse model of Alzheimer’s disease
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0170-z
Dihydromyricetin and Salvianolic acid B inhibit alpha-synuclein aggregation and enhance chaperone-mediated autophagy
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0159-7
Cognitive decline is related to high blood glucose levels in older Chinese adults with the ApoE ε3/ε3 genotype
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0151-2
Molecular targets for modulating the protein translation vital to proteostasis and neuron degeneration in Parkinson’s disease
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0145-0
DNA repair deficiency in neuropathogenesis: when all roads lead to mitochondria
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0156-x
Alzheimer’s in a dish – induced pluripotent stem cell-based disease modeling
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0161-0
No association between Parkinson disease and autoantibodies against NMDA-type glutamate receptors
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0153-0
Lower serum expression of miR-181c-5p is associated with increased plasma levels of amyloid-beta 1–40 and cerebral vulnerability in normal aging
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0174-8
Xenografting of human umbilical mesenchymal stem cells from Wharton’s jelly ameliorates mouse spinocerebellar ataxia type 1
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0166-8
NF-κB/c-Rel deficiency causes Parkinson’s disease-like prodromal symptoms and progressive pathology in mice
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0154-z
Clinical features and genetic spectrum in Chinese patients with recessive hereditary spastic paraplegia
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0157-9
Enriched gestation activates the IGF pathway to evoke embryo-adult benefits to prevent Alzheimer’s disease
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0149-9
Oral administration of the cannabigerol derivative VCE-003.2 promotes subventricular zone neurogenesis and protects against mutant huntingtin-induced neurodegeneration
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0148-x
Bee venom phospholipase A2 ameliorates amyloidogenesis and neuroinflammation through inhibition of signal transducer and activator of transcription-3 pathway in Tg2576 mice
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0167-7
Clinicopathologic characterization and abnormal autophagy of CSF1R-related leukoencephalopathy
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0171-y
Oscillation-specific nodal alterations in early to middle stages Parkinson’s disease
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0177-5
Transplantation of bone marrow derived macrophages reduces markers of neuropathology in an APP/PS1 mouse model
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0173-9
How to spot ocular abnormalities in progressive supranuclear palsy? A practical review
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0160-1
Kallikrein-related peptidases 6 and 10 are elevated in cerebrospinal fluid of patients with Alzheimer’s disease and associated with CSF-TAU and FDG-PET
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0168-6
Correction to: Better survival in female SOD1-mutant patients with ALS: a study of SOD1-related natural history
来源期刊：Translational NeurodegenerationDOI：10.1186/s40035-019-0150-3
