Skeletal Muscle

Skeletal Muscle

SKELET MUSCLE
影响因子:4.4
是否综述期刊:
是否预警:不在预警名单内
是否OA:
出版国家/地区:ENGLAND
出版社:BioMed Central
发刊时间:2011
发刊频率:
收录数据库:SCIE/Scopus收录/DOAJ开放期刊
ISSN:2044-5040

期刊介绍

The only open access journal in its field, Skeletal Muscle publishes novel, cutting-edge research and technological advancements that investigate the molecular mechanisms underlying the biology of skeletal muscle. Reflecting the breadth of research in this area, the journal welcomes manuscripts about the development, metabolism, the regulation of mass and function, aging, degeneration, dystrophy and regeneration of skeletal muscle, with an emphasis on understanding adult skeletal muscle, its maintenance, and its interactions with non-muscle cell types and regulatory modulators.Main areas of interest include:-differentiation of skeletal muscle-atrophy and hypertrophy of skeletal muscle-aging of skeletal muscle-regeneration and degeneration of skeletal muscle-biology of satellite and satellite-like cells-dystrophic degeneration of skeletal muscle-energy and glucose homeostasis in skeletal muscle-non-dystrophic genetic diseases of skeletal muscle, such as Spinal Muscular Atrophy and myopathies-maintenance of neuromuscular junctions-roles of ryanodine receptors and calcium signaling in skeletal muscle-roles of nuclear receptors in skeletal muscle-roles of GPCRs and GPCR signaling in skeletal muscle-other relevant aspects of skeletal muscle biology.In addition, articles on translational clinical studies that address molecular and cellular mechanisms of skeletal muscle will be published. Case reports are also encouraged for submission.Skeletal Muscle reflects the breadth of research on skeletal muscle and bridges gaps between diverse areas of science for example cardiac cell biology and neurobiology, which share common features with respect to cell differentiation, excitatory membranes, cell-cell communication, and maintenance. Suitable articles are model and mechanism-driven, and apply statistical principles where appropriate; purely descriptive studies are of lesser interest.
作为该领域唯一的开放获取期刊,《骨骼肌》发表了研究骨骼肌生物学基础分子机制的新颖、前沿的研究和技术进步。反映了这一领域研究的广度,该杂志欢迎有关骨骼肌的发育、代谢、质量和功能的调节、老化、退化、营养不良和再生的稿件,重点是了解成年骨骼肌、其维护及其与非肌肉细胞类型和调节调节剂的相互作用。主要感兴趣的领域包括:-骨骼肌的分化-骨骼肌的萎缩和肥大-骨骼肌的老化-骨骼肌的再生和退化-卫星和卫星样细胞的生物学-骨骼肌的营养不良性退化-骨骼肌的能量和葡萄糖稳态-骨骼肌的非营养不良性遗传疾病,如脊髓性肌萎缩和肌病-神经肌肉接头的维护-骨骼肌中兰尼定受体和钙信号的作用-骨骼肌中核受体的作用-骨骼肌中GPCR和GPCR信号的作用-骨骼肌生物学的其他相关方面。此外,还将发表关于骨骼肌分子和细胞机制的翻译临床研究骨骼肌反映了骨骼肌研究的广度,并弥合了不同科学领域之间的差距,例如心脏细胞生物学和神经生物学,它们在细胞分化、兴奋膜、细胞间通讯和维持方面具有共同特征。合适的文章是模型和机制驱动的,并在适当时应用统计学原理;纯粹描述性的研究兴趣不大。
年发文量 36
国人发稿量 6.26
国人发文占比 0.17%
自引率 -
平均录取率-
平均审稿周期 12 Weeks
版面费 US$2490
偏重研究方向 CELL BIOLOGY-
期刊官网 https://www.springer.com/journal/13395
投稿链接 https://www.editorialmanager.com/SKEM

期刊高被引文献

Culturing C2C12 myotubes on micromolded gelatin hydrogels accelerates myotube maturation
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0203-4
A GDF11/myostatin inhibitor, GDF11 propeptide-Fc, increases skeletal muscle mass and improves muscle strength in dystrophic mdx mice
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0197-y
Barium chloride injures myofibers through calcium-induced proteolysis with fragmentation of motor nerves and microvessels
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0213-2
IMB0901 inhibits muscle atrophy induced by cancer cachexia through MSTN signaling pathway
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0193-2
Estrogen signaling effects on muscle-specific immune responses through controlling the recruitment and function of macrophages and T cells
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0205-2
Automated image-analysis method for the quantification of fiber morphometry and fiber type population in human skeletal muscle
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0200-7
RNA-sequencing reveals altered skeletal muscle contraction, E3 ligases, autophagy, apoptosis, and chaperone expression in patients with critical illness myopathy
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0194-1
Late-onset megaconial myopathy in mice lacking group I Paks
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0191-4
The ancient sarcomeric myosins found in specialized muscles
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0192-3
NAD+ improves neuromuscular development in a zebrafish model of FKRP-associated dystroglycanopathy
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0206-1
Normal inflammation and regeneration of muscle following injury require osteopontin from both muscle and non-muscle cells
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0190-5
miR-1/206 downregulates splicing factor Srsf9 to promote C2C12 differentiation
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0211-4
Targeted ablation of the cellular inhibitor of apoptosis 1 (cIAP1) attenuates denervation-induced skeletal muscle atrophy
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0201-6
Muscle injury-induced hypoxia alters the proliferation and differentiation potentials of muscle resident stromal cells
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0202-5
miR-146a deficiency does not aggravate muscular dystrophy in mdx mice
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0207-0
mTORC2 affects the maintenance of the muscle stem cell pool
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0217-y
Regulation of murine skeletal muscle growth by STAT5B is age- and sex-specific
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0204-3
Exogenous expression of the glycosyltransferase LARGE1 restores α-dystroglycan matriglycan and laminin binding in rhabdomyosarcoma
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0195-0
Preclinical rationale for entinostat in embryonal rhabdomyosarcoma
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0198-x
Congenital myopathy with hanging big toe due to homozygous myopalladin (MYPN) mutation
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0199-9
Dilated cardiomyopathy-mediated heart failure induces a unique skeletal muscle myopathy with inflammation
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0189-y
LIM and cysteine-rich domains 1 (LMCD1) regulates skeletal muscle hypertrophy, calcium handling, and force
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0214-1
Muscle Gene Sets: a versatile methodological aid to functional genomics in the neuromuscular field
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0196-z
Two functional variants at 6p21.1 were associated with lean mass
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0212-3
Defective angiogenesis in CXCL12 mutant mice impairs skeletal muscle regeneration
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0210-5
Ibuprofen inhibited migration of skeletal muscle cells in association with downregulation of p130cas and CrkII expressions
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0208-z
Immunohistochemical phenotyping of T cells, granulocytes, and phagocytes in the muscle of cancer patients: association with radiologically defined muscle mass and gene expression
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0209-y
Development of a high-throughput screen to identify small molecule enhancers of sarcospan for the treatment of Duchenne muscular dystrophy
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0218-x
In memoriam: Susan Abmayr (1956–2019) – “What do we do? Whatever it takes!”
来源期刊:Skeletal MuscleDOI:10.1186/s13395-019-0215-0

质量指标占比

研究类文章占比 OA被引用占比 撤稿占比 出版后修正文章占比
100.00%100%-2.94%

相关指数

影响因子
影响因子
年发文量
自引率
Cite Score

预警情况

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时间 预警情况
2025年03月发布的2025版不在预警名单中
2024年02月发布的2024版不在预警名单中
2023年01月发布的2023版不在预警名单中
2021年12月发布的2021版不在预警名单中
2020年12月发布的2020版不在预警名单中
*来源:中科院《 国际期刊预警名单》

JCR分区

WOS分区等级:Q2区
版本 按学科 分区
WOS期刊SCI分区
WOS期刊SCI分区
WOS期刊SCI分区是指SCI官方(Web of Science)为每个学科内的期刊按照IF数值排 序,将期刊按照四等分的方法划分的Q1-Q4等级,Q1代表质量最高,即常说的1区期刊。
(2024-2025年最新版)
CELL BIOLOGY
Q2

中科院分区

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版本 大类学科 小类学科 Top期刊 综述期刊
2025年3月最新升级版
医学2区
CELL BIOLOGY 细胞生物学
2区
2023年12月升级版
医学2区
CELL BIOLOGY 细胞生物学
3区
2022年12月旧的升级版
医学2区
CELL BIOLOGY 细胞生物学
3区