The journal scope covers the entire spectrum of rheumatology research. We ensure that our articles are accessible to the widest possible audience.
Surgical and tissue engineering strategies for articular cartilage and meniscus repair
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0255-1
Shared and distinct mechanisms of fibrosis
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0322-7
Neuropsychiatric lupus: new mechanistic insights and future treatment directions
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-018-0156-8
T follicular helper cells and T follicular regulatory cells in rheumatic diseases
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0254-2
Systemic effects of IL-17 in inflammatory arthritis
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0243-5
The immunobiology of MIF: function, genetics and prospects for precision medicine
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0238-2
HLA associations in inflammatory arthritis: emerging mechanisms and clinical implications
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0219-5
The microbiome in SLE pathogenesis
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-018-0152-z
Chemokines in rheumatic diseases: pathogenic role and therapeutic implications
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0323-6
Rethinking articular cartilage regeneration based on a 250-year-old statement
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0278-7
Stratifying management of rheumatic disease for pregnancy and breastfeeding
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0240-8
Disease modification in OA — will we ever get there?
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0174-1
Involvement of the myeloid cell compartment in fibrogenesis and systemic sclerosis
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0212-z
Changing health behaviours in rheumatology: an introduction to behavioural economics
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0336-1
CAR T cells drive out B cells in SLE
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0214-x
No need for DMARD holiday before arthroplasty
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0199-5
Author Correction: Pathogenesis and therapeutic interventions for ANCA-associated vasculitis
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0168-z
Gut microbiota linked to kidney disease in SLE
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0196-8
Defective TFH cell checkpoint in SLE
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0182-1
Cholesterol test for T cells
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0195-9
Overcoming barriers to biosimilars in inflammatory arthritis
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0359-7
Could IL-6 inhibition prevent exercise-induced fat loss in RA?
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0189-7
Faulty mitochondrial DNA repair promotes inflammation in RA
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0303-x
Combined immunosuppression for ILD in myositis
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0328-1
Protecting against myocardial disease in SSc
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0299-2
Namilumab improves RA symptoms
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0231-9
Complement activation precedes classifiable SLE
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0310-y
Lung disease in sJIA has distinct features
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0298-3
Can personalized use of NSAIDs be a reality in the clinic?
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0225-7
Taking ABAT to OA
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0321-8
Do IL-7\u2009R+ innate cells orchestrate autoimmune pathology?
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0297-4
Mass cytometry markers for stratification of RA?
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0283-x
Should we use ultrasonography in the clinic to detect pSS?
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0305-8
Soluble CD83: a proresolving mediator in inflammatory arthritis?
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0224-8
New insights into myositis genetics
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0258-y
Next-generation analysis of synovial tissue architecture
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0357-9
Cartilage-on-a-chip to aid OA drug development
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0270-2
Belimumab slows organ damage progression
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0176-z
Hitting the right spot on CXCR4
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0287-6
Big data: the opportunity to think outside the discipline
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0292-9
Lactate rewires synovial T cells in RA
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0344-1
Controlling chondrocyte senescence
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0227-5
The choroid plexus: a cellular site of entry into the brain in NPSLE?
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0267-x
Gut microbiome linked with OA knee pain
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0346-z
Characterizing the BCR repertoire in immune-mediated diseases
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0339-y
Anti-TNF therapy reverses gut dysbiosis in AS
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0348-x
Lung biomarkers in SSc: prognostic or diagnostic?
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0177-y
LDHD mutation leads to hyperuricaemia and gout
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0347-y
Assisted reproduction less effective in RA
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0178-x
Has lupus anticoagulant testing had its day?
来源期刊：Nature Reviews RheumatologyDOI：10.1038/s41584-019-0218-6
