Vascular Pharmacology publishes papers, which contains results of all aspects of biology and pharmacology of the vascular system.Papers are encouraged in basic, translational and clinical aspects of Vascular Biology and Pharmacology, utilizing approaches ranging from molecular biology to integrative physiology. All papers are in English.The Journal publishes review articles which include vascular aspects of thrombosis, inflammation, cell signalling, atherosclerosis, and lipid metabolism.
血管药理学发表的论文包含血管系统生物学和药理学的所有方面的结果。鼓励发表血管生物学和药理学基础、转化和临床方面的论文，利用从分子生物学到综合生理学的方法。所有论文均以英语发表。该杂志发表的综述文章包括血栓形成、炎症、细胞信号传导、动脉粥样硬化和脂质代谢的血管方面。
LncRNAs in vascular biology and disease.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.01.003
Foam cell formation: A new target for fighting atherosclerosis and cardiovascular disease.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.08.002
Randomized controlled trial of rivaroxaban versus warfarin in the management of acute non-neoplastic portal vein thrombosis.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.05.002
Non-coding RNA regulation of endothelial and macrophage functions during atherosclerosis.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.03.001
MicroRNA-221-3p promotes pulmonary artery smooth muscle cells proliferation by targeting AXIN2 during pulmonary arterial hypertension.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2017.07.002
The function of miR-143, miR-145 and the MiR-143 host gene in cardiovascular development and disease
来源期刊：Vascular PharmacologyDOI：10.1016/j.vph.2018.11.006
Uric acid: from a biological advantage to a potential danger. A focus on cardiovascular effects.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.106565
Long non-coding RNAs in vascular biology and disease.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.03.003
Non-coding RNAs in vascular remodeling and restenosis.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.10.006
The function of long non-coding RNAs in vascular biology and disease.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.06.004
Linking diabetic vascular complications with LncRNAs.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.01.007
Non-coding RNAs in lipid metabolism.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.06.011
Pharmacological strategies to inhibit intra-plaque angiogenesis in atherosclerosis.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.06.014
Adenosine receptor agonists deepen the inhibition of platelet aggregation by P2Y12 antagonists.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.11.005
Beyond endothelial cells: Vascular endothelial growth factors in heart, vascular anomalies and placenta.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.10.005
Amorphous nano-selenium quantum dots improve endothelial dysfunction in rats and prevent atherosclerosis in mice through Na+/H+ exchanger 1 inhibition.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.01.005
Oxidative stress and vascular stiffness in hypertension: A renewed interest for antioxidant therapies?
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.03.004
Asiatic acid stabilizes cytoskeletal proteins and prevents TNF-α-induced disorganization of cell-cell junctions in human aortic endothelial cells.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.08.005
Vascular angiotensin AT1 receptor neuromodulation in fetal programming of hypertension.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.10.003
Matrix metalloproteinase (MMP)-2 activation by oxidative stress decreases aortic calponin-1 levels during hypertrophic remodeling in early hypertension.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.10.002
Cost effectiveness of lifelong therapy with PCSK9 inhibitors for lowering cardiovascular events in patients with stable coronary artery disease: Insights from the Ludwigshafen Risk and Cardiovascular Health cohort.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.106566
Alogliptin improves endothelial function by promoting autophagy in perivascular adipose tissue of obese mice through a GLP-1-dependent mechanism.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.11.003
Non-coding RNAs in aneurysmal aortopathy.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.06.008
Switching between P2Y12 antagonists - From bench to bedside.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.01.003
Modulation of the monocyte/macrophage system in heart failure by targeting heme oxygenase-1.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.08.011
Inflammation and myointimal hyperplasia. Correlation with hemodynamic forces.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2017.06.001
CRISPR links to long noncoding RNA function in mice: A practical approach.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.02.004
Treatment of cyclosporine induced hypertension: Results from a long-term observational study using different antihypertensive medications.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.06.012
Long non-coding RNA-SRA promotes neointimal hyperplasia and vascular smooth muscle cells proliferation via MEK-ERK-CREB pathway.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.02.005
Vitamin D levels and platelet reactivity in diabetic patients receiving dual antiplatelet therapy.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.106564
The endothelial status reflected by circulating endothelial cells, circulating endothelial progenitor cells and soluble thrombomodulin in patients with mild and resistant hypertension.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.12.005
Ex vivo microangioCT: Advances in microvascular imaging.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.09.003
HMGB1 enhances AGE-mediated VSMC proliferation via an increase in 5-LO-linked RAGE expression.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.04.001
Drug-induced hypertension: Know the problem to know how to deal with it.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.02.002
Role of amino acid metabolism in angiogenesis.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.11.001
Variety matters: Diverse functions of monocyte subtypes in vascular inflammation and atherogenesis.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.12.002
Microbiota are critical for vascular physiology: Germ-free status weakens contractility and induces sex-specific vascular remodeling in mice.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.106633
SNF472, a novel anti-crystallization agent, inhibits induced calcification in an in vitro model of human aortic valve calcification.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.106583
Stox1 induced the proliferation and cell cycle arrest in pulmonary artery smooth muscle cells via AKT signaling pathway.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.106568
Effects of sildenafil on maximum walking time in patients with arterial claudication: The ARTERIOFIL study.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.05.003
Long non-coding RNAs: A crucial part of the vasculature puzzle.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.02.008
Everolimus depletes plaque macrophages, abolishes intraplaque neovascularization and improves survival in mice with advanced atherosclerosis.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.12.004
Rapid effects of aldosterone on platelets, coagulation, and fibrinolysis lead to experimental thrombosis augmentation.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.106598
Elevated luteinizing hormone contributes to atherosclerosis formation by inhibiting nitric oxide synthesis via PI3K/Akt pathway.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.106582
Lipid core nanoparticles as vehicle for docetaxel reduces atherosclerotic lesion, inflammation, cell death and proliferation in an atherosclerosis rabbit model.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.02.003
LIM-only protein FHL2 attenuates inflammation in vascular smooth muscle cells through inhibition of the NFκB pathway.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.106634
GLP-1-based therapies to boost autophagy in cardiometabolic patients: From experimental evidence to clinical trials.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.03.003
Atorvastatin enhances endothelial adherens junctions through promoting VE-PTP gene transcription and reducing VE-cadherin-Y731 phosphorylation.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.06.003
Antiretroviral drug-induced endothelial dysfunction is improved by dual PPARα/γ stimulation in obesity.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2019.106577
Lessons learned from a lncRNA odyssey for two genes with vascular functions, DLL4 and TIE1.
来源期刊：Vascular pharmacologyDOI：10.1016/j.vph.2018.06.010
