Nature Reviews Nephrology aims to be the premier source of reviews and commentaries for the scientific communities we serve. We strive to publish articles that are authoritative, accessible and enhanced with clearly understandable figures, tables and other display items. We aim to provide an unparalleled service to authors, referees and readers, and to maximize the usefulness and impact of each article. The journal publishes Research Highlights, News & Views, Comments, Reviews, Perspectives and Consensus Statements relevant to nephrologists and basic science researchers, with our broad scope ensuring that the work we publish reaches the widest possible audience.
Nature Reviews Nephrology旨在成为我们所服务的科学界的评论和评论的首要来源。我们努力发表的文章是权威的，可访问的，并加强了清晰易懂的数字，表格和其他显示项目。我们的目标是为作者、审稿人和读者提供无与伦比的服务，并最大限度地提高每篇文章的实用性和影响力。该杂志发表与肾脏病学家和基础科学研究者相关的研究亮点、新闻与观点、评论、综述、观点和共识声明，我们的广泛范围确保我们发表的工作达到尽可能广泛的受众。
Cellular signalling by primary cilia in development, organ function and disease
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0116-9
Endothelium structure and function in kidney health and disease
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-018-0098-z
The role of inflammasomes in kidney disease
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0158-z
The case for uric acid-lowering treatment in patients with hyperuricaemia and CKD
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0174-z
The genetic changes of Wilms tumour
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0112-0
Immunity, microbiota and kidney disease
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0118-7
Measured and estimated glomerular filtration rate: current status and future directions
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0191-y
Mechanisms of phosphate transport
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0159-y
Immunology of the ageing kidney
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0185-9
Molecular nephrology: types of acute tubular injury
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0184-x
Mechanisms of haemolysis-induced kidney injury
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0181-0
Sex and the kidneys: current understanding and research opportunities
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0208-6
Reappraising the spectrum of AKI and hepatorenal syndrome in patients with cirrhosis
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0218-4
The (pro)renin receptor in health and disease
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0160-5
Artificial intelligence to predict AKI: is it a breakthrough?
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0203-y
Progress in the understanding of polycystic kidney disease
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-018-0108-1
Considerations for SGLT2 inhibitor use in post-transplantation diabetes
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0173-0
Kidney disease trials for the 21st century: innovations in design and conduct
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0212-x
Artificial intelligence in nephropathology
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0220-x
Synthetic lethality between loss of CDK4/6 activity and VHL inactivation
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0225-5
The structure of Na+-K+-Cl− cotransporter 1
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0204-x
Type IV collagen and diabetic kidney disease
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0229-1
PC2–mitochondria interactions in PKD
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0162-3
AHR activation by uraemic solutes
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0230-8
Lipids mediate podocyte damage
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0196-6
Tolerogenic cell therapy for glomerulonephritis
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0206-8
Disparities between trial cohorts and real-life patients
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0202-z
A molecular imaging approach for the early, real-time diagnosis of acute kidney injury
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0165-0
Single-cell analysis identifies key pathways and prognostic markers in lupus nephritis
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0164-1
STING activation by cytoplasmic mtDNA triggers renal inflammation and fibrosis
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0211-y
A materials approach for kidney organoids
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0139-2
Albuminuria: a target for clinical trials in kidney disease?
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0123-x
A novel approach to targeting TGFβ1
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0219-3
Nephropathic autoantigens in the spectrum of lupus nephritis
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0168-x
Not your usual parietal cell
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0141-8
Some nephron progenitors fail to commit
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0122-y
Growing mouse kidneys in rats
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0127-6
Toxins linked to chronic interstitial nephritis in agricultural communities
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0239-z
MENTOR heralds a new era of therapy for membranous nephropathy
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0200-1
CNVs in CAKUT
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0115-x
A novel SGLT1-mediated mechanism of hyperglycaemia-induced hyperfiltration
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0146-3
Histocompatibility: minor differences have a major impact
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0175-y
The genetic architecture of blood pressure
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0117-8
PAX8: a candidate oncogene in RCC
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0205-9
CUBN variants uncouple proteinuria from kidney function
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0242-4
Recreating, expanding and using nephron progenitor populations
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0238-0
VE-PTP phosphatase: a new target for diabetic kidney disease
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0147-2
Nitric oxide protects against AKI by reprogramming metabolism
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0113-z
Tubuloids for modelling kidney disease
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0138-3
Kidney miR-33 controls fatty acid oxidation
来源期刊：Nature Reviews NephrologyDOI：10.1038/s41581-019-0228-2
