JOURNAL OF ENDOCRINOLOGY

JOURNAL OF ENDOCRINOLOGY

J ENDOCRINOL
影响因子:3.9
是否综述期刊:
是否预警:不在预警名单内
是否OA:
出版国家/地区:ENGLAND
出版社:BioScientifica Ltd.
发刊时间:1939
发刊频率:Monthly
收录数据库:SCIE/Scopus收录
ISSN:0022-0795

期刊介绍

Journal of Endocrinology is a leading global journal that publishes original research articles, reviews and science guidelines. Its focus is on endocrine physiology and metabolism, including hormone secretion; hormone action; biological effects. The journal publishes basic and translational studies at the organ, tissue and whole organism level.
《内分泌学杂志》是一本全球领先的杂志,发表原创研究文章、评论和科学指南。它的重点是内分泌生理学和代谢,包括激素分泌;激素作用;生物效应。该杂志发表器官、组织和整个生物体水平的基础和转化研究。
年发文量 76
国人发稿量 5.84
国人发文占比 0.08%
自引率 -
平均录取率0
平均审稿周期 偏慢,4-8周
版面费 -
偏重研究方向 医学-内分泌学与代谢
期刊官网 JOURNAL OF ENDOCRINOLOGY
投稿链接 http://www.endocrinology-journals.org/site/misc/submit-online.xhtml

期刊高被引文献

Epigenetic responses and the developmental origins of health and disease.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0009
Inhibitors of apoptosis protect the ovarian reserve from cyclophosphamide.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0370
Nutritional adversity, sex and reproduction: 30 years of DOHaD and what have we learned?
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0048
Epidemiological evidence for the developmental origins of health and disease: effects of prenatal undernutrition in humans.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0683
Leptin resensitisation: a reversion of leptin-resistant states.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0606
Developmental programming of the HPA axis and related behaviours: epigenetic mechanisms.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0057
Central growth hormone signaling is not required for the timing of puberty.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0242
Nrf2 represses the onset of type 1 diabetes in non-obese diabetic mice.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0355
Impact of exenatide on mitochondrial lipid metabolism in mice with nonalcoholic steatohepatitis.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0007
Maternal vitamin D deficiency and developmental origins of health and disease (DOHaD).
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0541
Terpenes, hormones, and life: isoprene rule revisited.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0084
Effect of exercise and butyrate supplementation on microbiota composition and lipid metabolism.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0122
Developmental pathways and programming of diabetes: epidemiological aspects.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0680
Mice lacking adenosine 2A receptor reveal increased severity of MCD-induced NASH.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0198
Metformin improves ovarian insulin signaling alterations caused by fetal programming.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0520
High glucose up-regulates osteopontin expression by FoxO1 activation in macrophage.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0594
Thyroid hormone signalling and consequences for cardiac development.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0704
The glucocorticoid receptor in osteoprogenitors regulates bone mass and marrow fat.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0230
The depot-specific and essential roles of CBP/p300 in regulating adipose plasticity.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0361
Telmisartan prevents development of obesity and normalizes hypothalamic lipid droplets.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0319
Voluntary exercise is motivated by ghrelin, possibly related to the central reward circuit
来源期刊:The Journal of EndocrinologyDOI:10.1530/JOE-19-0213
Chronic predator stress in female mice reduces primordial follicle numbers: implications for the role of ghrelin.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0109
Identification of circular RNAs in the immature and mature rat anterior pituitary.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0540
Antenatal dexamethasone treatment transiently alters diastolic function in the mouse fetal heart
来源期刊:The Journal of EndocrinologyDOI:10.1530/JOE-18-0666
Mechanism of SEMA3G knockdown-mediated attenuation of high-fat diet-induced obesity.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0029
High glucose alters fetal rat islet transcriptome and induces progeny islet dysfunction.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0493
Vagal afferents contribute to sympathoexcitation-driven metabolic dysfunctions.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0623
Comparing methods to normalize insulin secretion shows the process may not be needed.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0542
L- and T-type calcium channels control aldosterone production from human adrenals.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0259
SRT1720 retards renal fibrosis via inhibition of HIF1α /GLUT1 in diabetic nephropathy.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0536
Testosterone inhibits aneurysm formation and vascular inflammation in male mice.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0646
Anti-insulin resistance effects of salidroside through mitochondrial quality control.
来源期刊:The Journal of endocrinologyDOI:10.1530/joe-19-0393
Prenatal ethanol exposure induces susceptibility to premature ovarian insufficiency.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0063
BMP7 improves insulin signal transduction in the liver.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0693
Role of ovarian sympathetic nerves and cholinergic local system during cold stress.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0125
CaV1.2 and CaV1.3 voltage-gated L-type Ca2+ channels in rat white fat adipocytes.
来源期刊:The Journal of endocrinologyDOI:10.1530/joe-19-0493
Role of neutrophil extracellular traps in chronic kidney injury induced by bisphenol-A.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0608
Impaired LH surge amplitude in gonadotrope-specific progesterone receptor knockout mice.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0013
Cholestasis-associated glucocorticoid overexposure does not increase atherogenesis.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0079
Adhesion G-protein coupled receptor, GPR56, is required for Müllerian duct development in the chick.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0419
Monomethylfumarate protects against ovariectomy-related changes in body composition.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0691
Vincamine as a GPR40 agonist improves glucose homeostasis in type 2 diabetic mice.
来源期刊:The Journal of endocrinologyDOI:10.1530/joe-18-0432
Hyperinsulinemia restrains endometrial angiogenesis during decidualization in mice and human models.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0127
Hematopoiesis in aged female mice devoid of thyroid hormone receptors.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0339
Thermoneutrality improves skeletal impairment in adult Prader-Willi syndrome mice.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0279
Baicalin inhibits recruitment of GATA1 to the HSD3B2 promoter and reverses hyperandrogenism of PCOS.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-18-0678
Selenoprotein S regulates adipogenesis through IRE1α-XBP1 pathway.
来源期刊:The Journal of endocrinologyDOI:10.1530/joe-19-0292
Glucagon regulates hepatic mitochondrial function and biogenesis through FOXO1.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0081
Forced internal desynchrony induces cardiometabolic alterations in adult rats.
来源期刊:The Journal of endocrinologyDOI:10.1530/JOE-19-0026
Inhibition of vascular adventitial remodeling by netrin-1 in diabetic rats.
来源期刊:The Journal of endocrinologyDOI:10.1530/joe-19-0258

质量指标占比

研究类文章占比 OA被引用占比 撤稿占比 出版后修正文章占比
51.32%25.23%0.79%-

相关指数

影响因子
影响因子
年发文量
自引率
Cite Score

预警情况

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时间 预警情况
2025年03月发布的2025版不在预警名单中
2024年02月发布的2024版不在预警名单中
2023年01月发布的2023版不在预警名单中
2021年12月发布的2021版不在预警名单中
2020年12月发布的2020版不在预警名单中
*来源:中科院《 国际期刊预警名单》

JCR分区

WOS分区等级:Q2区
版本 按学科 分区
WOS期刊SCI分区
WOS期刊SCI分区
WOS期刊SCI分区是指SCI官方(Web of Science)为每个学科内的期刊按照IF数值排 序,将期刊按照四等分的方法划分的Q1-Q4等级,Q1代表质量最高,即常说的1区期刊。
(2024-2025年最新版)
ENDOCRINOLOGY & METABOLISM
Q2

中科院分区

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版本 大类学科 小类学科 Top期刊 综述期刊
2025年3月最新升级版
医学3区
ENDOCRINOLOGY & METABOLISM 内分泌学与代谢
4区
2023年12月升级版
医学3区
ENDOCRINOLOGY & METABOLISM 内分泌学与代谢
3区
2022年12月旧的升级版
医学2区
ENDOCRINOLOGY & METABOLISM 内分泌学与代谢
3区