Kidney International (KI) is the official journal of the International Society of Nephrology. Under the editorial leadership of Dr. Detlef O. Schlöndorff (USA), KI is one of the most cited journals in nephrology and widely regarded as the world's premier journal on the development and consequences of kidney disease.
《肾脏国际》（KI）是国际肾脏病学会的官方期刊。在Detlef O.Schlöndorff（美国），KI是肾脏病学中被引用最多的期刊之一，被广泛认为是世界上关于肾脏疾病发展和后果的首要期刊。
Comparison of organ donation and transplantation rates between opt-out and opt-in systems.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.01.036
Dialysis initiation, modality choice, access, and prescription: conclusions from a Kidney Disease: Improving Global Outcomes (KDIGO) Controversies Conference.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.01.017
Mineralocorticoid receptor antagonists and kidney diseases: pathophysiological basis.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.02.030
Arterial “inflammaging” drives vascular calcification in children on dialysis
来源期刊：Kidney internationalDOI：10.1016/j.kint.2018.12.014
Wnt/β-catenin links oxidative stress to podocyte injury and proteinuria.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2018.10.032
Big science and big data in nephrology.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2018.11.048
Multifactorial functions of the inflammasome component NLRP3 in pathogenesis of chronic kidney diseases.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.01.014
Biological variation of measured and estimated glomerular filtration rate in patients with chronic kidney disease.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.02.021
Chronic kidney disease and valvular heart disease: conclusions from a Kidney Disease: Improving Global Outcomes (KDIGO) Controversies Conference.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.06.025
Safety and cardiovascular efficacy of spironolactone in dialysis-dependent ESRD (SPin-D): a randomized, placebo-controlled, multiple dosage trial.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2018.08.034
Organelle crosstalk in the kidney.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2018.11.035
Endothelin receptor-A mediates degradation of the glomerular endothelial surface layer via pathologic crosstalk between activated podocytes and glomerular endothelial cells.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.05.007
Mononuclear phagocytes orchestrate prolyl hydroxylase inhibition-mediated renoprotection in chronic tubulointerstitial nephritis.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.02.016
ATF6α downregulation of PPARα promotes lipotoxicity-induced tubulointerstitial fibrosis.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2018.09.023
Aberrant DNA methylation of mTOR pathway genes promotes inflammatory activation of immune cells in diabetic kidney disease.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.02.020
Prolyl hydroxylase inhibition protects the kidneys from ischemia via upregulation of glycogen storage.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.10.020
CX3CL1-CX3CR1 interaction mediates macrophage-mesothelial cross talk and promotes peritoneal fibrosis.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2018.12.030
Whole exome sequencing identified ATP6V1C2 as a novel candidate gene for recessive distal renal tubular acidosis.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.09.026
Increased production and reduced urinary buffering of acid in uric acid stone formers is ameliorated by pioglitazone.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2018.11.024
Mechanisms of GLP-1 receptor-independent renoprotective effects of the dipeptidyl peptidase type 4 inhibitor linagliptin in GLP-1 receptor knockout mice with 5/6 nephrectomy.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.01.010
Detection and characterization of mosaicism in autosomal dominant polycystic kidney disease.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.08.038
High-throughput sequencing contributes to the diagnosis of tubulopathies and familial hypercalcemia hypocalciuria in adults.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.08.027
Activation of fibroblastic reticular cells in kidney lymph node during crescentic glomerulonephritis.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2018.08.040
Dexmedetomidine reduces norepinephrine requirements and preserves renal oxygenation and function in ovine septic acute kidney injury.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.06.013
Renal globotriaosylceramide facilitates tubular albumin absorption and its inhibition protects against acute kidney injury.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.02.010
Tissue hypoxia, inflammation, and loss of glomerular filtration rate in human atherosclerotic renovascular disease.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2018.11.039
Mutations in KIRREL1, a slit diaphragm component, cause steroid-resistant nephrotic syndrome.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.06.016
The IgG-degrading enzyme of Streptococcus pyogenes causes rapid clearance of anti-glomerular basement membrane antibodies in patients with refractory anti-glomerular basement membrane disease.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.06.019
DAMPening sterile inflammation of the kidney.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2018.12.007
A propensity score matched analysis shows no adverse effect of early steroid withdrawal in non-diabetic kidney transplant recipients with and without glomerulonephritis.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.02.041
Expanding opportunities and emerging challenges: broadening the scope of genetic testing in nephrology.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2018.12.032
Apolipoprotein L1 (APOL1) risk variant toxicity depends on the haplotype background.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.07.010
Transgenic zebrafish modeling low-molecular-weight proteinuria and lysosomal storage diseases.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.11.016
Chronic kidney diseases in agricultural communities: report from a workshop.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.06.024
Age-related changes in DNA methylation affect renal histology and post-transplant fibrosis.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.06.018
Inhibition of the H3K4 methyltransferase MLL1/WDR5 complex attenuates renal senescence in ischemia reperfusion mice by reduction of p16INK4a.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.06.021
Bioplasmonic paper-based assay for perilipin-2 non-invasively detects renal cancer.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.08.020
Improving glomerular filtration rate estimation.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.01.016
Genetic characterization of early renal changes in a novel mouse model of diabetic kidney disease.
来源期刊：Kidney internationalDOI：10.1016/J.KINT.2019.04.031
The zinc fingers and homeoboxes 2 protein ZHX2 and its interacting proteins regulate upstream pathways in podocyte diseases.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.11.011
Avian erythroblastosis virus E26 oncogene homolog-1 (ETS-1) plays a role in renal microvascular pathophysiology in the Dahl salt-sensitive rat.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.09.025
How to sharpen a novel sword from AKI basic research.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2018.09.017
Clinical features of genetically confirmed patients with primary hyperoxaluria identified by clinical indication versus familial screening.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.11.023
Shiga toxin-producing Escherichia coli-associated hemolytic uremic syndrome in solid organ transplant recipients.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.08.024
Noncanonical immunomodulatory activity of complement regulator C4BP(β-) limits the development of lupus nephritis.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.10.016
Blood pressure modifies outcomes in patients with stage 3 to 5 chronic kidney disease.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.10.021
Ferric citrate: cardio- and renoprotective in chronic kidney disease?
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.09.004
An unexpected role of steroid on podocytes: from zebrafish to human nephrotic syndrome?
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.01.044
The impact of epilepsy and its treatment in patients with end-stage renal disease.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.06.010
ATHENA: wisdom and warfare in defining the role of de novo mTOR inhibition in kidney transplantation.
来源期刊：Kidney internationalDOI：10.1016/j.kint.2019.04.020
