The Journal of Molecular and Cellular Cardiology publishes work advancing knowledge of the mechanisms responsible for both normal and diseased cardiovascular function. To this end papers are published in all relevant areas. These include (but are not limited to): structural biology; genetics; proteomics; morphology; stem cells; molecular biology; metabolism; biophysics; bioengineering; computational modeling and systems analysis; electrophysiology; pharmacology and physiology. Papers are encouraged with both basic and translational approaches. The journal is directed not only to basic scientists but also to clinical cardiologists who wish to follow the rapidly advancing frontiers of basic knowledge of the heart and circulation.
《分子和细胞心脏病学杂志》发表了有关正常和疾病心血管功能机制的前沿知识。为此目的，在所有相关领域发表了论文。包括（但不限于）：结构生物学;蛋白质组学;形态学;干细胞;分子生物学;新陈代谢;生物物理学;生物工程;计算建模和系统分析;电生理学;药理学和生理学。论文鼓励采用基础和翻译两种方法。该杂志不仅面向基础科学家，也面向希望了解心脏和循环基础知识快速发展前沿的临床心脏病专家。
Circular RNA Ttc3 regulates cardiac function after myocardial infarction by sponging miR-15b.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.03.007
Mitochondrial LonP1 protects cardiomyocytes from ischemia/reperfusion injury in vivo.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2018.12.017
Gut microbe-derived metabolite trimethylamine N-oxide accelerates fibroblast-myofibroblast differentiation and induces cardiac fibrosis.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.07.004
p53 plays a crucial role in endothelial dysfunction associated with hyperglycemia and ischemia.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.02.010
DNA methyltransferase 1 and Krüppel-like factor 4 axis regulates macrophage inflammation and atherosclerosis.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.01.009
Cardiac repair and the putative role of stem cells.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.01.022
Cardiac-specific deficiency of the mitochondrial calcium uniporter augments fatty acid oxidation and functional reserve.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2018.12.019
Enhancement of human iPSC-derived cardiomyocyte maturation by chemical conditioning in a 3D environment.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.10.001
A short history of the development of mathematical models of cardiac mechanics
来源期刊：Journal of Molecular and Cellular CardiologyDOI：10.1016/j.yjmcc.2018.11.015
Oxidative stress contributes to vascular calcification in patients with chronic kidney disease.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.12.006
Ablation of the calpain-targeted site in cardiac myosin binding protein-C is cardioprotective during ischemia-reperfusion injury.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.03.006
Heparanase protects the heart against chemical or ischemia/reperfusion injury.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.04.008
Programmed death-ligand 1 triggers PASMCs pyroptosis and pulmonary vascular fibrosis in pulmonary hypertension.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.10.008
Early inhibition of endothelial retinoid uptake upon myocardial infarction restores cardiac function and prevents cell, tissue, and animal death.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2018.11.012
Crucial role of NLRP3 inflammasome in a murine model of Kawasaki disease.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.11.158
Pseudo-bipolar spindle formation and cell division in postnatal binucleated cardiomyocytes.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.07.005
Cardiac arrhythmias after renal I/R depend on IL-1β.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.04.025
HDAC inhibition as a therapeutic strategy in myocardial ischemia/reperfusion injury.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.02.013
Myocardial maladaptation to pressure overload in CB2 receptor-deficient mice.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.06.003
Exosomal non-coding RNAs (Exo-ncRNAs) in cardiovascular health.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.09.016
Phosphorylation of Cx43 residue Y313 by Src contributes to blocking the interaction with Drebrin and disassembling gap junctions.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2018.11.008
Dysregulation of IL-33/ST2 signaling and myocardial periarteriolar fibrosis.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.01.018
Requisite roles of LOX-1, JNK, and arginase in diabetes-induced endothelial vasodilator dysfunction of porcine coronary arterioles.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.04.015
Multiple roles of KLF15 in the heart: Underlying mechanisms and therapeutic implications.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.01.024
Activation of the calcium-sensing receptor in human valvular interstitial cells promotes calcification.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.01.021
Heterotypic docking compatibility of human connexin37 with other vascular connexins.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2018.12.013
Deficiency of NONO is associated with impaired cardiac function and fibrosis in mice.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.10.004
Smooth muscle-specific LKB1 deletion exaggerates angiotensin II-induced abdominal aortic aneurysm in mice.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.03.021
Sam68 impedes the recovery of arterial injury by augmenting inflammatory response.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.10.003
Hypoplastic left heart syndrome: From bedside to bench and back.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.08.005
Inhibition of DRG-TRPV1 upregulation in myocardial ischemia contributes to exogenous cardioprotection.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.12.003
Protective transcriptional mechanisms in cardiomyocytes and cardiac fibroblasts.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.04.023
In vitro and in vivo cardioprotective and metabolic efficacy of vitamin E TPGS/Apelin.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.12.001
MIR148A family regulates cardiomyocyte differentiation of human embryonic stem cells by inhibiting the DLL1-mediated NOTCH signaling pathway.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.06.014
Genetic and functional implications of an exonic TRIM55 variant in heart failure.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.12.008
Inhibition of Kir2.1 channel-induced depolarization promotes cell biological activity and differentiation by modulating autophagy in late endothelial progenitor cells.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2018.11.005
Mitochondrial thioredoxin-2 maintains HCN4 expression and prevents oxidative stress-mediated sick sinus syndrome.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.10.009
Large scale, unbiased analysis of elementary calcium signaling events in cardiac myocytes.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.08.004
Conversion of human cardiac progenitor cells into cardiac pacemaker-like cells.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.09.015
Shp2 in myocytes is essential for cardiovascular and neointima development.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.09.014
Intrafibrillar and perinuclear mitochondrial heterogeneity in adult cardiac myocytes.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.08.013
Homocysteine accelerates atherosclerosis by inhibiting scavenger receptor class B member1 via DNMT3b/SP1 pathway.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.11.145
The evolutionarily conserved C-terminal peptide of troponin I is an independently configured regulatory structure to function as a myofilament Ca2+-desensitizer.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.09.002
Angiotensin peptide synthesis and cyclic nucleotide modulation in sympathetic stellate ganglia
来源期刊：Journal of Molecular and Cellular CardiologyDOI：10.1016/j.yjmcc.2019.11.157
Kv4.3 expression abrogates and reverses norepinephrine-induced myocyte hypertrophy by CaMKII inhibition.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2018.11.011
Tachycardia-induced CD44/NOX4 signaling is involved in the development of atrial remodeling.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.08.006
Cytoplasmic nucleic acid-based XNAs directly enhance live cardiac cell function by a Ca2+ cycling-independent mechanism via the sarcomere.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.02.016
The lymphocyte adapter protein: A negative regulator of myocardial ischemia/reperfusion injury.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.07.003
Cardiac expression of the microsomal triglyceride transport protein protects the heart function during ischemia.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.09.003
Novel role of mitochondrial GTPases 1 in pathological cardiac hypertrophy.
来源期刊：Journal of molecular and cellular cardiologyDOI：10.1016/j.yjmcc.2019.01.025
