Welcome and thank you for considering submitting your work to Cell Metabolism. Established in 2005, Cell Metabolism is the top research journal dedicated to publishing novel, impactful papers spanning basic to clinical metabolic research. We are interested in original research addressing the molecular mechanisms underlying physiological homeostasis and what goes awry in disease.The journal's mission is to provide the metabolic community a forum for the exchange of ideas and concepts and to promote cross-disciplinary research and collaboration. In line with this, we aim to publish work that is not only of exceptional significance within its field, but also of interest to researchers outside the immediate area, promoting idea cross-pollination. Cell Metabolism's scope includes diabetes, obesity, pancreatic beta cell function, adipose tissue biology (white, brown, and beige), lipid metabolism, cardiovascular biology, immunometabolism, bone homeostasis, aging and stress responses, sarcopenia, intermediary metabolism and cancer, cachexia, neuronal control of metabolism and metabolic derangements causing neurodegeneration, circadian biology, stem cell energetics, exercise metabolism, the biology of mitochondria and other metabolic organelles such as peroxisomes and the ER, etc.For original research articles, different formats are available such as "Research Article," "Short Article," "Clinical and Translational Report," and "Resource."For non-research (front matter) articles, the formats include "Letter," "Essay," "Minireview," "Review," and "Perspective."
欢迎并感谢您考虑将您的工作提交给细胞代谢。2005年，细胞代谢是顶级研究期刊，致力于发表新颖的，有影响力的论文，涵盖基础到临床代谢研究。我们对研究生理稳态的分子机制以及疾病的问题感兴趣。该杂志的使命是为代谢界提供一个交流思想和概念的论坛，并促进跨学科研究和合作。为此，我们的目标是发表不仅在其领域内具有特殊意义的工作，而且对附近地区以外的研究人员也感兴趣的工作，以促进idea异花授粉。细胞代谢的范围包括糖尿病，肥胖，胰腺 β 细胞功能，脂肪组织生物学(白色，棕色和米色)，脂质代谢，心血管生物学，免疫代谢，骨稳态，衰老和应激反应，肌肉减少，中介代谢和癌症，恶病质，神经元控制代谢和引起神经变性的代谢紊乱，昼夜节律生物学，干细胞能量学，运动代谢，线粒体生物学和其他代谢细胞器，如过氧化物酶体和ER等。对于原创研究文章，可以使用不同的格式，例如“研究文章”，“短文”，“临床和翻译报告”和“资源”。对于非研究(前题)文章，格式包括“信函”，“论文”，“迷你评论”，“评论”和“透视”。
Caloric Restriction Mimetics against Age-Associated Disease: Targets, Mechanisms, and Therapeutic Potential.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.01.018
GDF15 Provides an Endocrine Signal of Nutritional Stress in Mice and Humans
来源期刊：Cell MetabolismDOI：10.1016/j.cmet.2018.12.016
Exercise-Induced Changes in Visceral Adipose Tissue Mass Are Regulated by IL-6 Signaling: A Randomized Controlled Trial.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2018.12.007
The Mouse Microbiome Is Required for Sex-Specific Diurnal Rhythms of Gene Expression and Metabolism
来源期刊：Cell MetabolismDOI：10.1016/j.cmet.2018.09.023
Lymphocyte-Derived Exosomal MicroRNAs Promote Pancreatic β Cell Death and May Contribute to Type 1 Diabetes Development.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2018.09.011
Extracellular Vesicle-Contained eNAMPT Delays Aging and Extends Lifespan in Mice.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.05.015
Cholesterol Induces CD8+ T Cell Exhaustion in the Tumor Microenvironment.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.04.002
NAD+ in Brain Aging and Neurodegenerative Disorders.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.09.001
Genetic Analysis Reveals AMPK Is Required to Support Tumor Growth in Murine Kras-Dependent Lung Cancer Models.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2018.10.005
A Map of Human Type 1 Diabetes Progression by Imaging Mass Cytometry.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2018.11.014
Choline Uptake and Metabolism Modulate Macrophage IL-1β and IL-18 Production.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.03.011
Preserving Insulin Secretion in Diabetes by Inhibiting VDAC1 Overexpression and Surface Translocation in β Cells
来源期刊：Cell MetabolismDOI：10.1016/j.cmet.2018.09.008
Mitochondrial Damage and Activation of the STING Pathway Lead to Renal Inflammation and Fibrosis.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.08.003
Mediation of the Acute Stress Response by the Skeleton.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.08.012
Lactate Buildup at the Site of Chronic Inflammation Promotes Disease by Inducing CD4+ T Cell Metabolic Rewiring
来源期刊：Cell MetabolismDOI：10.1016/j.cmet.2019.10.004
Non-Canonical Control of Neuronal Energy Status by the Na+ Pump.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2018.11.005
Epithelial-Mesenchymal Transition Directs Stem Cell Polarity via Regulation of Mitofusin.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2018.11.004
Cell Clustering Promotes a Metabolic Switch that Supports Metastatic Colonization
来源期刊：Cell MetabolismDOI：10.1016/j.cmet.2019.07.014
Acyl-CoA-Binding Protein Drives Glioblastoma Tumorigenesis by Sustaining Fatty Acid Oxidation.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.04.004
Deubiquitinases Maintain Protein Homeostasis and Survival of Cancer Cells upon Glutathione Depletion.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.01.020
Microbial Metabolite Signaling Is Required for Systemic Iron Homeostasis.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.10.005
Acyl-CoA-Binding Protein Is a Lipogenic Factor that Triggers Food Intake and Obesity.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.07.010
Neurocognitive and Hormonal Correlates of Voluntary Weight Loss in Humans.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2018.09.024
Calcium Signaling Controls Pathogenic Th17 Cell-Mediated Inflammation by Regulating Mitochondrial Function.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.01.019
Transient Receptor Potential V Channels Are Essential for Glucose Sensing by Aldolase and AMPK
来源期刊：Cell MetabolismDOI：10.1016/j.cmet.2019.05.018
Aerobic Plus Resistance Exercise in Obese Older Adults Improves Muscle Protein Synthesis and Preserves Myocellular Quality Despite Weight Loss.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.06.008
Revisiting How the Brain Senses Glucose-And Why.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2018.11.001
Targeting Peripheral CB1 Receptors Reduces Ethanol Intake via a Gut-Brain Axis.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.04.012
Gut-Proglucagon-Derived Peptides Are Essential for Regulating Glucose Homeostasis in Mice.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.08.009
Tanycytes Regulate Lipid Homeostasis by Sensing Free Fatty Acids and Signaling to Key Hypothalamic Neuronal Populations via FGF21 Secretion.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.08.004
Acylglycerol Kinase Maintains Metabolic State and Immune Responses of CD8+ T Cells.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.05.016
Fructose-1,6-Bisphosphatase 2 Inhibits Sarcoma Progression by Restraining Mitochondrial Biogenesis.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.10.012
Patient-Driven Diabetes Care of the Future in the Technology Era.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2018.09.005
The DAPA-HF Trial: A Momentous Victory in the War against Heart Failure.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.10.008
AMPK-Mediated Lysosome Biogenesis in Lung Cancer Growth.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2018.12.011
United They Stand, Divided They Fall.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.09.008
Disruption of Acetyl-Lysine Turnover in Muscle Mitochondria Promotes Insulin Resistance and Redox Stress without Overt Respiratory Dysfunction.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.11.003
The Untapped Opportunity and Challenge of Immunometabolism: A New Paradigm for Drug Discovery.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.11.014
Insulin-Driven PI3K-AKT Signaling in the Hepatocyte Is Mediated by Redundant PI3Kα and PI3Kβ Activities and Is Promoted by RAS.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.03.010
Regulatory T Cells under the Mercy of Mitochondria.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.01.012
Host Control of Tumor Feeding: Autophagy Holds the Key.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.01.009
Adipose Tissue B Cells Come of Age: The AABs of Fat Inflammation.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.11.007
New Insights into the Regulation of Leptin Gene Expression.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.04.005
A Gut Check Explains Improved Glucose Metabolism after Surgery.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.10.002
Gut T Cells Feast on GLP-1 to Modulate Cardiometabolic Disease.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.03.002
Metabolic Adaptation Fuels Lymph Node Metastasis.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.03.006
Mitochondrial DNA Inheritance in Humans: Mix, Match, and Survival of the Fittest.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.07.009
Acyl-CoA-Binding Protein Fuels Gliomagenesis.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.07.007
The Transsulfuration Pathway Makes, the Tumor Takes.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.10.009
TMEM2 Modulates ER Stress in a Non-canonical Manner.
来源期刊：Cell metabolismDOI：10.1016/j.cmet.2019.11.008
